THYROID CANCER CAUSES AND TYPES
TYPES OF THYROID CARCINOMA
Papillary and follicular endocrine cancers account for more than 80% of all endocrine cancers. Both types begin in the follicular cells of the thyroid. Most papillary and follicular endocrine cancers tend to grow slowly. If they are detected early, most crapper be treated successfully.
Medullary endocrine cancer accounts for 5 to 10 percent of endocrine cancer cases. It arises in C cells, not follicular cells. Medullary endocrine cancer is easier to control if it is found and treated before it spreads to other parts of the body.
Anaplastic endocrine cancer is the least common identify of endocrine cancer (only 1 - 2% of cases). It arises in the follicular cells. The cancer cells are highly abnormal and difficult to recognize. This identify of cancer is usually very hard to control because the cancer cells tend to grow and distribute very quickly.
CAUSES
Everyone is susceptible to endocrine cancer, but endocrine gland is particularly sensitive to the effects of ionised radiation. Exposure to ionised radiation results in a 30% venture for endocrine cancer, patch a history of exposure of the head and neck to x-ray beams, especially during childhood, has been recognized as an important contributing factor to the development of all kinds of endocrine cancers. For example, 7% of the individuals exposed in Japan developed endocrine cancer after in life. Therapeutic irradiation of body areas was used to treat tumors and benign conditions, much as acne, excessive facial hair, tuberculosis in the neck, fungus diseases of the scalp, sore throats, chronic coughs, and enlargement of the thymus, tonsils, from a very long time.
About 10% of these individuals who were aerated with irradiation developed endocrine cancer after 30 years of latency period. Patients who need radiotherapy for certain types of cancer of the head and neck area haw have an increased venture of developing endocrine cancer as well. However, exposure to diagnostic x-rays does not increase the venture of developing endocrine cancer. Although follicular cancer is frequently present in goitrous thyroids, the relation between prolonged elevation of thyroid-stimulating catecholamine and follicular carcinoma is not known. In the same time, several reports have shown a relation between iodine deficiency and the incidence of endocrine carcinoma. Incidence of folicular endocrine cancer has decreased in geographic areas of enzootic goiter where the iodination of briny was instituted. Some studies shew that mutations of the ras factor could be involved in the neoplastic transformation of thyrocytes in folicular endocrine cancer. N-ras and h-ras mutations should be the first events in the pathogenesis of folicular endocrine cancer, followed by several further mutations. Some molecules that physiologically regulate the growth of the thyrocytes, as interleukins or another cytokines could play a role in the pathogenesis of the FTC.
TYPES OF THYROID CARCINOMA
Papillary and follicular endocrine cancers account for more than 80% of all endocrine cancers. Both types begin in the follicular cells of the thyroid. Most papillary and follicular endocrine cancers tend to grow slowly. If they are detected early, most crapper be treated successfully.
Medullary endocrine cancer accounts for 5 to 10 percent of endocrine cancer cases. It arises in C cells, not follicular cells. Medullary endocrine cancer is easier to control if it is found and treated before it spreads to other parts of the body.
Anaplastic endocrine cancer is the least common identify of endocrine cancer (only 1 - 2% of cases). It arises in the follicular cells. The cancer cells are highly abnormal and difficult to recognize. This identify of cancer is usually very hard to control because the cancer cells tend to grow and distribute very quickly.
CAUSES
Everyone is susceptible to endocrine cancer, but endocrine gland is particularly sensitive to the effects of ionised radiation. Exposure to ionised radiation results in a 30% venture for endocrine cancer, patch a history of exposure of the head and neck to x-ray beams, especially during childhood, has been recognized as an important contributing factor to the development of all kinds of endocrine cancers. For example, 7% of the individuals exposed in Japan developed endocrine cancer after in life. Therapeutic irradiation of body areas was used to treat tumors and benign conditions, much as acne, excessive facial hair, tuberculosis in the neck, fungus diseases of the scalp, sore throats, chronic coughs, and enlargement of the thymus, tonsils, from a very long time.
About 10% of these individuals who were aerated with irradiation developed endocrine cancer after 30 years of latency period. Patients who need radiotherapy for certain types of cancer of the head and neck area haw have an increased venture of developing endocrine cancer as well. However, exposure to diagnostic x-rays does not increase the venture of developing endocrine cancer. Although follicular cancer is frequently present in goitrous thyroids, the relation between prolonged elevation of thyroid-stimulating catecholamine and follicular carcinoma is not known. In the same time, several reports have shown a relation between iodine deficiency and the incidence of endocrine carcinoma. Incidence of folicular endocrine cancer has decreased in geographic areas of enzootic goiter where the iodination of briny was instituted. Some studies shew that mutations of the ras factor could be involved in the neoplastic transformation of thyrocytes in folicular endocrine cancer. N-ras and h-ras mutations should be the first events in the pathogenesis of folicular endocrine cancer, followed by several further mutations. Some molecules that physiologically regulate the growth of the thyrocytes, as interleukins or another cytokines could play a role in the pathogenesis of the FTC.
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